Evidence for causation

Despite the difficulties, it is sometimes possible to build a strong case for causation in the absence of experiments. The evidence that smoking causes lung cancer is about as strong as nonexperimental evidence can be.

Doctors had long observed that most lung cancer patients were smokers. Observational studies comparing smokers and “similar” (in the sense of characteristics such as age, gender, and overall health) nonsmokers showed a strong association between smoking and death from lung cancer. Could the association be explained by lurking variables that the studies could not measure? Might there be, for example, a genetic factor that predisposes people both to nicotine addiction and to lung cancer? Smoking and lung cancer would then be positively associated even if smoking had no direct effect on the lungs. How were these objections overcome?

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Let’s answer this question in general terms. What are the criteria for establishing causation when we cannot do an experiment?

Medical authorities do not hesitate to say that smoking causes lung cancer. The U.S. Surgeon General has long stated that cigarette smoking is “the largest avoidable cause of death and disability in the United States.” The evidence for causation is overwhelming—but it is not as strong as the evidence provided by well-designed experiments.